How Does Someone Go Into A Coma

How Does Someone Go Into A Coma – In Saturday’s attack outside a supermarket in Tucson, U.S. Rep. In the case of Gabrielle Giffords, who suffered a severe brain injury from a gunshot wound that killed six people and injured 13 others, doctors sometimes put her in a coma. This effective shutdown of brain function naturally only occurs in extreme cases of infection, so why do doctors try to replicate it in patients, as they have the congresswoman, who has still suffering from head injuries and other problems?

The answer lies in modern anesthesia technology, which about 60,000 patients go through every day. Review the article published in the December 30, 2010 issue

How Does Someone Go Into A Coma

This is what doctors look for in a true drug-induced coma, which is often induced by exposure to a cold environment using similar drugs or extreme hypothermia to completely cut off blood flow and function. aorta. Stopping work can give the brain time to recover without the body making drastic changes by cutting off blood flow to damaged areas. To learn more about these medically induced comas and the reasons doctors use them,

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Basically, what happens in a drug-induced coma is that you take the drug and use it until you see a certain pattern on the machine that tracks the patient’s brain waves, which is the EEG [electroencephalogram]. Brain-injured patients who are unconscious have a similar pattern. If that pattern exists, then you feel comfortable knowing that the patient is drug-induced unconsciousness. You do this to protect your brain.

If you have a brain injury, what happens is a major change in brain metabolism. You may have areas that don’t have enough blood flow. The idea is, “Let me reduce the amount of energy required by the different areas of the brain.” If I can do that, the brain heals and the inflammation goes down, maybe the areas that were at risk can be protected.

But the main thing about a drug-induced coma, unlike a coma, is that it can be reversed. If you do this to someone with a normal brain, they will recover as soon as you stop the drugs.

In general, the main effect of these drugs outside the brain is to lower blood pressure. So people who try to do this are given many other drugs to keep the blood pressure high and the heart pumping properly. On the one hand, you protect the brain and on the other hand, not all parts of the brain get the blood they need.

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If you do this for a long time, the drugs can build up and take time to get out of your system. As long as you know these things, you can help someone get through such a time.

It really depends on the injury, whether it’s a brain injury or a concussion. One patient spent six months in a [drug-induced coma]. It is clearly the end of the distribution.

It depends on how the person progresses and the type of injury. What neurologists or [intensive care unit doctors] do is try to get them out as quickly as possible. In cases like Gifford’s it is swollen. If they see that the swelling has gone down, they can try to relieve the coma to see if it comes back and see how their work is.

A drug like propofol, we use it every day in the operating room. It is probably the most widely used drug in all anesthesia. Often, every day, when patients receive general anesthesia, the entire state is a reversible coma. It varies in values.

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Usually the body does not decide to go into a coma. Coma is a complete cessation of brain activity. It is often the result of severe trauma, brain injury, drug overdose, stroke, severe insult. There is no natural analogue for [medication-induced coma].

It is difficult to solve, because if you overdo these things you are already facing a very serious situation. If there are effects, it is very difficult to distinguish whether they are the effects of drug-induced coma. People who do this are very careful to observe and monitor. They try their best to use this option when necessary. For other uses, see Disambiguation. Not to be confused with vowels or vowels. “Comas” leads here. For other uses, see Disambiguation.

Coma is a prolonged, deep state of unconsciousness in which a person cannot wake up, does not respond normally to painful stimuli, light or sound, does not have a normal sleep cycle,’ and he does not initiate voluntary actions.

A person may experience breathing and circulatory problems due to the body’s inability to maintain normal bodily functions. People who are unconscious often need extensive medical attention to maintain their health and prevent complications such as pneumonia or bleeding.

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It can also be defined as a Glasgow Coma Scale (GCS) ≤ 8 lasting ≥ 6 hours.

To preserve the fallen mind, the elements of awakening and awareness must be preserved. Awake describes the state of awareness, while awareness is related to the qualitative aspects of the functions involved in the cortex, including cognitive abilities such as attention, historical perspective, vivid memory, language and task performance. Actions, the temporal and spatial pattern and validity of justice. .

From a neurological perspective, awareness is maintained by the activation of the cerebral cortex (gray matter that forms the outer part of the brain) and the reticular activating system (RAS), a structure located in the brain stem. .

The word ‘coma’, from the Greek κῶμα koma, meaning deep sleep, was already used in the Hippocratic Corpus (Epidemica) and later in Gaul (second century AD). Later it was not used much in popular literature until the middle of the 17th century. The term reappears in the influential De Anima Grossum (1672) by Thomas Willis (1621-1675), where lethargy (pathological sleep), ‘coma’ (deep sleep), carus (senselessness) and apoplexy (where carus can. turn and those in space with white matter). The word carus also comes from the Greek, where it can be found at the root of many words meaning soporific or sleepy. It can still be found in the root of the word “carotid”. Thomas Sidham (1624–89) mentioned the word “coma” in many cases of fever (Sidham, 1685).

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Under certain conditions the use of certain drugs can destroy or weaken the synaptic activity in the increased reticular activating system (ARAS) and prevent the system from working properly to stimulate the brain.

Side effects of medications, including abnormal heart rate and blood pressure, as well as abnormal breathing and sweating, can also indirectly impair ARAS function and lead to coma. Because a large proportion of comatose patients are caused by drug poisoning, hospitals begin to examine all comatose patients with the vestibular-ocular reflex, by observing the size of the pupils and eye movements. (See review below.)

The second most common cause of coma, which accounts for about 25% of cases, is lack of oxygen, which is often the result of cardiac arrest.

The central nervous system (CNS) needs a lot of oxygen for its nerves. Lack of oxygen in the brain, also known as hypoxia, reduces sodium and calcium outside of neurons and increases intracellular calcium, disrupting neuronal communication.

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Lack of oxygen in the brain also leads to a decrease in ATP and cell damage due to cytoskeletal damage and nitric oxide production.

During a stroke, blood flow to a part of the brain is blocked or restricted. An ischemic stroke, a brain hemorrhage, or a brain tumor can cause a blockage of blood flow. Lack of blood supply to the brain cells prevents oxygen from reaching the neurons and as a result, the cells are damaged and die. When brain cells die, brain cells are damaged, which can affect ARAS activity and lead to seizures and coma.

Abnormal sugar levels and many other biological problems. Furthermore, studies show that 1 in 8 people with a brain injury develop a coma.

Cardiovascular causes of coma include cardiac arrest, heart attack, heart attack, heart failure, acute shock, cardiac arrest, myocarditis and pericarditis. Pulmonary arrest is the only lung condition that causes coma, but many different lung conditions can decrease mental status but not lead to coma.

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Other causes of coma include severe or persistent falls, kidney failure, liver failure, hyperglycemia, hypoglycemia, and diseases affecting the brain, such as meningitis and cephalitis.

Damage to one or both of the cerebral cortex or the reticular activating system (RAS) is enough to put a person into a coma.

The cerebral cortex is made up of gray matter that contains the nuclei of neurons, while the interior of the brain is made up of white matter and contains the axons of neurons.

White matter is responsible for vision, transmission of sensory information through the thalamic pathway, and many other neurological functions, including complex thinking.

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RAS, Mme

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